Calcific neurocysticercosis and epileptogenesis

Theodore E. Nash, O. H. Del Brutto, J. A. Butman, T. Corona, A. Delgado-Escueta, R. M. Duron, C. A.W. Evans, R. H. Gilman, A. E. Gonzalez, J. A. Loeb, M. T. Medina, S. Pietsch-Escueta, E. J. Pretell, O. M. Takayanagui, W. Theodore, V. C.W. Tsang, H. H. Garcia

Research output: Contribution to journalScientific review

174 Scopus citations

Abstract

Neurocysticercosis is responsible for increased rates of seizures and epilepsy in endemic regions. The most common form of the disease, chronic calcific neurocysticercosis, is the end result of the host's inflammatory response to the larval cysticercus of Taenia solium. There is increasing evidence indicating that calcific cysticercosis is not clinically inactive but a cause of seizures or focal symptoms in this population. Perilesional edema is at times also present around implicated calcified foci. A better understanding of the natural history, frequency, epidemiology, and pathophysiology of calcific cysticercosis and associated disease manifestations is needed to define its importance, treatment, and prevention.
Original languageAmerican English
Pages (from-to)1934-1938
Number of pages5
JournalNeurology
DOIs
StatePublished - 8 Jun 2004

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    Nash, T. E., Del Brutto, O. H., Butman, J. A., Corona, T., Delgado-Escueta, A., Duron, R. M., Evans, C. A. W., Gilman, R. H., Gonzalez, A. E., Loeb, J. A., Medina, M. T., Pietsch-Escueta, S., Pretell, E. J., Takayanagui, O. M., Theodore, W., Tsang, V. C. W., & Garcia, H. H. (2004). Calcific neurocysticercosis and epileptogenesis. Neurology, 1934-1938. https://doi.org/10.1212/01.WNL.0000129481.12067.06